By Chakrapani (Ed.)
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Additional resources for Chandamama (1955) July
Interpretation of the relevance of genotoxicity becomes much more complex in the case of naturally occurring constituents and ubiquitous environmental contaminants, particularly as many foods also contain constituents that are able to modulate genotoxic activity. Many other systems have been proposed as alternatives to animals for assessing toxicity, and some of those most applicable to food chemicals are reviewed elsewhere in this book. Approaches include computer modelling, expert systems, structure-activity relationships and many different in vitro assays, ranging from undifferentiated cell lines, with basal cytotoxicity endpoints, to complex culture systems with sensitive measures of cell function as endpoints.
All available animal, in vitro and human toxicity data are assessed in order to identify the toxicological consequences that might be expected to result from human exposure and, if possible, to postulate a biological mechanism. Having identified a toxicological endpoint, it is then necessary to characterize the dose-response relationship. There are two general models used to characterize dose-response relationships. 2). e. that no dose has no effect but even the smallest dose has some, albeit small, effect).
Whilst some aspects are relatively well established, for the most part risk analysis is in its early stages of development and is likely to change dramatically over the next few decades - particularly in the field of food chemical safety. Much of the science of risk analysis as it is applied to food chemicals is drawn from other disciplines, particularly engineering. The principles are broadly similar regardless of the application, although there may be significant differences in detail. One important difference is that engineering risk analysis can often be built upon experience - accident and failure rates associated with different operations within a particular industry, for example.